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Repeated blockade of NMDA receptors during adolescence impairs reversal learning and disrupts GABAergic interneurons in rat medial prefrontal cortex
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Although GPX4, FSP1, and DHODH all contribute to ferroptosis suppression through the maintenance of reduced ubiquinol, DHODH represents the principal enzymatic defense against ferroptosis within mitochondria [214,215,216], while GPX4 and FSP1 predominantly act in non-mitochondrial membranes [217]
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